NURS 6512 Assignment 3 Digital Clinical Experience Comprehensive (Head-to-Toe) Physical Assessment IN<\/strong><\/h2>\n\u00a0<\/strong><\/p>\nOBJECTIVE DATA<\/strong><\/h3>\n<\/h3>\n
Vital signs:<\/strong> Respiration- 15<\/p>\nTemp- 37.2 C<\/p>\n
Heartrate – 78<\/p>\n
SpO2- 99%<\/p>\n
\n- Height: 170 cm<\/li>\n
- Weight: 84 kg<\/li>\n
- BMI: 29.0<\/li>\n
- Blood Glucose: 100<\/li>\n
- RR: 15<\/li>\n
- HR: 78<\/li>\n
- BP:128 \/ 82<\/li>\n
- Pulse Ox: 99%<\/li>\n
- Temperature: 99.0 F<\/li>\n<\/ul>\n
General:<\/strong> Jones is alert oriented, seated upright on examination table, and is in no distress. She is well-nourished, developed, and dressed appropriately with good hygiene.<\/p>\nHEENT: <\/strong>Head is normocephalic, atraumatic. Eyes bilateral with equal hair distribution on lashes and eyebrows. No lesions on lids, no edema or ptosis. Pink conjunctiva, white sclera, PERRLA bilaterally, intact extraocular eye movements, and no nystagmus. Mild retinopathic changes on right. Left fundus with sharp disc margins, no hemorrhages. Snellen: 20\/20 right eye, 20\/20 left eye with corrective lenses. TMs intact and pearly gray bilaterally, positive light reflex. Whispered words heard bilaterally. Frontal and maxillary sinuses nontender to palpation. Nasal mucosa moist and pink, septum midline. Oral mucosa moist without ulcerations or lesions, uvula rises midline on phonation. Gag reflex intact. Dentition without evidence of caries or infection.<\/p>\nNeck: <\/strong>Thyroid smooth without nodules, no goiter. No lymphadenopathy.<\/p>\nChest\/Lungs: <\/strong>Lung sounds clear and voice is present in all areas. Spanish symmetrically. Chest anterior and posterior normal upon inspection. fremitus equal bilaterally. Chest is symmetric with respiration, clear to auscultation bilaterally without cough or wheeze. Resonant to percussion throughout. In office spirometry: FVC 3.91 L, FEV1\/FVC ratio 80.56%.<\/p>\nHeart\/Peripheral Vascular: <\/strong>Pirated 2 + with no thrill or bruit bilaterally. PMI non-discplaced. S1 and S2 only regular rhythm. No bruit in aorta or any other arteries. Capillary refill is less than 3 seconds in fingers and toes no edema is present. Heart rate is regular, S1, S2, without murmurs, gallops, or rubs. Bilateral carotids equal bilaterally without bruit. PMI at the midclavicular line, 5th intercostal space, no heaves, lifts, or thrills. Bilateral peripheral pulses equal bilaterally, capillary refill less than 3 seconds. No peripheral edema.<\/p>\nAbdomen: <\/strong>bowel sounds are normal in all quadrants. moves bowels regularly. Abdomen is soft with no Masses. liver is one centimeter below the right costal margin. Quadrants are tympanic and spleen is Not dull in sound. Kidney is not palpable no masses are present<\/p>\n <\/p>\n
Abdomen protuberant, symmetric, no visible masses, scars, or lesions, coarse hair from pubis to umbilicus. Bowel sounds are normoactive in all four quadrants. Tympanic throughout to percussion. No tenderness or guarding to palpation. No organomegaly. No CVA tenderness.<\/p>\n
Genital\/Rectal: <\/strong>–<\/h2>\nMusculoskeletal: <\/strong>Range of motion in all areas of full or muscle strength or 5 out of 5 no CVA tenderness. DTR 2+. Strength 5\/5 bilateral upper and lower extremities, without swelling, masses, or deformity and with full range of motion. No pain with movement.<\/p>\n\u00a0<\/strong><\/p>\nNeurological: <\/strong>for the feet especially left foot area. Patient is able to sense position of body fingers and toes. Graphesthesia normal sense. Patient is oriented to time person and place. Heel to Shin normal. Normal graphesthesia, stereognosis, and rapid alternating movements bilaterally. Tests of cerebellar function normal. DTRs 2+ and equal bilaterally in upper and lower extremities. Decreased sensation to monofilament in bilateral plantar surfaces.<\/p>\nSkin: <\/strong>Acne is present on the face. Skin is normal. Norwegians or abnormalities in the nails. Old scar is present on the left shin.<\/p>\nScattered pustules on face and facial hair on upper lip, acanthosis nigricans on posterior neck. Nails free of ridges or abnormalities.<\/p>\n
\u00a0<\/strong><\/p>\nDiagnostic results: <\/strong>None<\/h2>\n\u00a0<\/strong><\/p>\nASSESSMENT: <\/strong>Jones is a 28-year-old female that has come today for her pre-employment assessment. She appears well dressed and responsive. She is diabetic and asthmatic, which are controlled. She uses corrective lenses. She has normal sleeping cycle. She engages in active physical activity and has dietary modifications for diabetes control. She monitors her blood glucose levels on a daily basis. She also monitors her peak flow to track asthma and uses albuterol inhaler to manage its symptoms. She denies any current acute health problems. \u00a0<\/strong><\/p>\n\u00a0<\/strong><\/p>\nPLAN: <\/strong>This section is not required for the assignments in this course (NURS 6512), but will be required for future courses.<\/em><\/p>\nS.<\/u><\/strong><\/h3>\nCC<\/strong>: \u201cI have a headache around my forehead.\u201d<\/p>\nHPI<\/strong>: J.K.L is a 40-year-old African American female who presents with a complaint of a headache across her forehead for a week. The headache is squeezing and feels like pressure behind the eyes. It is non-radiating. The headache is constant and varies in severity ranging from 2\/10 at its best to 8\/10 at its worst. It is usually worse in the morning and while bending. Acetaminophen reduces the severity of the headache to 4\/10 and occasionally 2\/10. It is associated with fever, postnasal drip, nasal congestion, sneezing, and occasional non-productive cough. She takes Sudafed HCL 120 mg every 12 hours to obtain some relief. The symptoms have significantly impaired her concentration at work and made her feel very tired. Finally, she reports a head cold three weeks ago.<\/p>\nCurrent Medications<\/strong>: Pseudoephedrine 120 mg BID for nasal congestion and acetaminophen for headaches.<\/p>\nAllergies: <\/strong>She has no known food and drug allergies.<\/p>\nPast Medical History<\/strong>: During her last visit to the primary care physician 2 months ago, she was noted to be prehypertensive and was advised on lifestyle modifications. No prior hospitalization. No previous surgeries or blood transfusions.<\/p>\nSocial History<\/strong>: She is married with two children both alive and well. She works as a secretary Her husband is a college teacher. She neither drinks alcohol nor smokes tobacco. She does not use marijuana or other illicit drugs. She strictly adheres to dietary advice from her nutritionist and she exercises regularly. Denies caffeine intake.<\/p>\nFamily History<\/strong>: Father alive aged 60 years and with hypertension while her mother is 58 years old alive and well. Her brother and sister are 35 and 20 years old respectively, alive and well. Her paternal grandfather died at the age of 80 years due to a heart attack while her paternal grandmother is 78 years and is hypertensive. Her maternal grandfather is 77 years with a history of type 2 diabetes and high cholesterol while her maternal grandmother died at the age of 70 years due to a stroke. No family history of malignancies, mental illness, asthma, sickle cell, or diabetes.<\/p>\nROS<\/strong>:<\/h2>\nGENERAL: Reports fatigue and occasional fever. Denies weight loss, night sweats, and chills.<\/p>\n
HEENT: \u00a0Reports headaches, nasal congestion, post nasal drip, and sneezing. No blurring of vision, visual loss, hearing loss, tinnitus, nose bleeds, ear pain, mouth sores, or sore throat.<\/p>\n
SKIN: \u00a0no skin lesion or rashes. No abnormal pigmentation.<\/p>\n
CARDIOVASCULAR: Negative for palpitations, chest pain, paroxysmal nocturnal dyspnea, and peripheral limb edema.<\/p>\n
RESPIRATORY: \u00a0Occasional non-productive cough. No difficulty in breathing, dyspnea, or orthopnea.<\/p>\n
GASTROINTESTINAL: Reports loss of appetite and occasional nausea and vomiting. Denies change in bowel habits, abdominal pain, or distention.<\/p>\n
GENITOURINARY:\u00a0No frequency, dysuria, nocturia, and polyuria. No vaginal itchiness or abnormal vaginal discharge.<\/p>\n
NEUROLOGICAL: Reports headache. Denies dizziness, lightheadedness, numbness, tingling, loss of sensation, syncope, and convulsion.<\/p>\n
MUSCULOSKELETAL: No muscle pain, joint pains, muscle weakness, or muscle swelling.<\/p>\n
HEMATOLOGIC: \u00a0No anemia, easy bruising, or bleeding.<\/p>\n
LYMPHATICS: Normal lymph nodes<\/p>\n
PSYCHIATRIC: \u00a0Denies anxiety, depression, suicidal ideations, or hallucinations.<\/p>\n
ENDOCRINOLOGIC: Denies heat or cold intolerance, polyphagia, and polydipsia.<\/p>\n
ALLERGIES: \u00a0Reports no allergies.<\/p>\n
O.<\/u><\/strong><\/h3>\nPhysical exam<\/strong>:<\/h2>\nVITAL SIGNS: BP 125\/78 mmHg, HR 88 b\/min, Temp 99. 8 F, RR 20 b\/min, saturation 95% on room air, Height 168 cm, weight 76 Kg. Pain level 5\/10<\/p>\n
GENERAL: A middle-aged African-American female, well kempt, not in any form of respiratory distress but slight discomfort. Maintains eye contact, coherent speech, and a stable mood. Well-hydrated and nourished. No palmar or conjunctival pallor, jaundice, central or peripheral cyanosis, cervical or inguinal lymphadenopathy, and peripheral limb edema.<\/p>\n
HEENT: Normocephalic and atraumatic head. Non-tender scalp. Bilateral eyes with pink conjunctiva and white sclera. Pupils equally and bilaterally reacting to light, no ptosis or lid edema. Normal extraocular movements. Bilateral ears present, no impaction or skin lesions, tympanic membrane pearly grey bilaterally, and positive white reflex. Both nares are present and are discharging mucus, midline nasal septum, and pink and soft nasal mucosa. Tender maxillary and frontal sinus. Moist and pink oral mucosa, no oral lesions or ulceration. Normal dentition and teeth alignment.<\/p>\n
NECK: Soft neck. The trachea is central. Full range of motion, non-tender, no cervical lymphadenopathy, and no thyroid enlargement.<\/p>\n
CARDIOVASCULAR: Regular heart rate. Normoactive precordium. Point of maximal impulse in the 5th<\/sup> intercostal space in the midclavicular line. S1 and S2 head, no murmurs, thrills, gallops, rubs, or heaves.<\/p>\nRESPIRATORY: Symmetrical chest that moves with respiration. No scars or skin lesions. Equal chest expansion and equal tactile fremitus bilaterally. Equal air entry, vesicular breath sounds, no wheezes, and crackles, and equal vocal fremitus in all lung zones.<\/p>\n
NEUROLOGICAL: GCS 15\/15, oriented to time, place, and person, intact short-term and long-term memory, good concentration, and a clear coherent speech. Cranial nerves 1 to 12 intact. Normotonic across all joints, normal bulk, and power 5\/5 across all muscle groups in upper and lower extremities, deep tendon reflexes 2+ and equal bilaterally in upper and lower limbs. Intact monofilament sensation across all dermatomes, good bowel, and bladder function. No spinal tenderness, normal gait, coordination, graphesthesia, and stereognosis. Normal finger nose, heel to the shin, and rapid alternating movements tests.<\/p>\n
Diagnostic results<\/strong>:<\/h2>\nJ.K.L appears to have an inflammatory\/infectious condition. Consequently, complete blood count and inflammatory markers particularly CRP and ESR are paramount. Similarly, bacterial or fungal cultures obtained endoscopically or by direct sinus aspiration are required to identify the possible pathogen. Additionally, a skin prick test is essential to exclude allergic rhinitis. Imaging modalities principally Sinus CT and MRI are recommended to evaluate for rhinosinusitis and intraorbital or intracranial involvement.<\/p>\n
<\/p>\n
A<\/u><\/strong>.<\/u><\/h3>\nDifferential Diagnoses<\/strong><\/h2>\nAcute Sinusitis- <\/strong>refers to the inflammation of sinuses lasting less than 4 weeks (DeBoer & Kwon, 2022). The condition is more common in females and particularly during early fall to early spring (DeBoer & Kwon, 2022). It is most commonly caused by viral infection following a common cold although bacteria and fungi are not uncommon etiologies. J.K.L presents with clinical features that are typical of acute sinusitis including fatigue, fever, headache, facial pain, and pressure worse on bending (DeBoer & Kwon, 2022). Maxillary sinuses and frontal sinuses appear to be the affected sinuses in her as evidenced by pain around the forehead and tenderness of the maxillary and frontal sinuses (DeBoer & Kwon, 2022).<\/p>\nRhinitis- <\/strong>Refers to the inflammation of the nasal mucosa. J.K.L presents with clinical manifestations suggestive of rhinitis including sneezing, nasal congestion, postnasal drip, and rhinorrhea (Liva et al., 2021). Similarly, she reports a \u201chead cold\u201d three weeks ago. Rhinitis is mostly caused by an upper respiratory infection or type 1 hypersensitivity reaction (Liva et al., 2021). However, an upper respiratory tract infection is likely the cause in her case.<\/p>\nCluster headache- <\/strong>Cluster headache is a type of primary headache that is usually unilateral retro-orbital and characterized by sharp and stabbing pain (Goadsby et al., 2018). Cluster headache may present with symptoms of lacrimation, nasal congestion, rhinorrhea, ptosis, or miosis (Goadsby et al., 2018). However, it is unlikely the diagnosis in her as cluster headache usually lasts for a brief period. Similarly, cluster headaches mostly awake the patient at night.<\/p>\nMigraine headache- <\/strong>Migraine headache is another type of primary headache that may be preceded with or without aura. It is usually pulsating and moderate to severe (Pescador Ruschel & O, 2022). It is common in young women. However, it is unlikely the diagnosis as migraines last 4 to 72 hours if untreated and are typically associated with nausea, vomiting, photophobia, and phonophobia (Pescador Ruschel & O, 2022).<\/p>\nRebound headache<\/strong>– Commonly referred to as medication overuse headache. Rebound headache predominantly occurs in individuals with primary headaches who overuse analgesia (Micieli & Robblee, 2018). Rebound headaches are more common in females and individuals less than 50 years. Drugs precipitating this headache include barbiturates, acetaminophen, opioids, ergotamine, and triptans (Micieli & Robblee, 2018). However, this is an unlikely diagnosis in J.K.L as a diagnosis of primary headache hasn\u2019t been established.<\/p>\n <\/p>\n