In this exercise, you will complete a 5-essay type question Knowledge Check to gauge your understanding of this module\u2019s content. \u202f<\/span>\u00a0<\/span><\/p>\n GASTROINTESTINAL AND HEPATOBILIARY DISORDERS<\/span>\u00a0<\/span><\/a><\/p>\n Possible topics covered in this Knowledge Check include:<\/span>\u00a0<\/span><\/p>\n Be sure to review the Learning Resources before completing this activity.<\/span> WEEKLY RESOURCES<\/span><\/b><\/a>\u00a0<\/span><\/p>\n Complete\u202f<\/span><\/b>the Knowledge Check By Day 7 of Week 5.<\/span>\u00a0<\/span><\/p>\n Attempt History<\/span>\u00a0<\/span><\/p>\n Score for this quiz:\u202f<\/span>18.7<\/span><\/b>\u202fout of 20<\/span>\u00a0<\/span><\/p>\n Submitted Apr 2 at 8:51am<\/span>\u00a0<\/span><\/p>\n This attempt took 8,805 minutes.<\/span>\u00a0<\/span><\/p>\n \u202f<\/span><\/p>\n A 65-year-old female comes to the clinic with a complaint of abdominal pain in the epigastric area. The pain has been persistent for two weeks. \u202fThe pain described as burning, non-radiating and worse after meals. Denies N&V, weight loss or obvious bleeding. She admits to frequent belching with bloating.\u202f\u202f<\/span>\u00a0<\/span><\/p>\n PMH:\u202f seasonal allergies with Chronic Sinusitis, positive for osteoarthritis,\u202f<\/span>\u00a0<\/span><\/p>\n Meds: Claritin 10 mg po daily, ibuprofen 400-600 mg po prn pain\u202f<\/span>\u00a0<\/span><\/p>\n Family Hx-non\u202fcontributary\u202f\u202f<\/span>\u00a0<\/span><\/p>\n Social history: Separated recently pending divorce; stressful situation with trying to manage two homes. Works as a Legal Assistant at a local law firm. She has 35 PPY of smoking, drinks 1-2 glasses of wine a day, and 6-7 cups of coffee per day. She\u202fdenies illicit drug use, vaping or unprotected sexual encounters.\u202f\u202f<\/span>\u00a0<\/span><\/p>\n Breath test in the office revealed + urease.\u202f<\/span>\u00a0<\/span><\/p>\n The healthcare provider suspects the client has peptic ulcer disease.<\/span>\u00a0<\/span><\/p>\n 1.<\/span><\/i>\u202f\u202f\u202f\u202f\u202f<\/span><\/i>Explain what contributed to the development from this patient\u2019s history of PUD?<\/span><\/i>\u00a0<\/span><\/p>\n \u00a0<\/span><\/p>\n Your Answer:<\/span>\u00a0<\/span><\/p>\n Peptic ulcer disease (PUD) occurs following impairment of the mucosal defenses, which leaves them incapable of protecting the epithelium from the effects of acid and pepsin.\u202f The development of PUD is associated primarily with bacterial infection with H. pylori and NSAIDs.\u202f NSAIDs like diclofenac and ibuprofen break down the stomach mucosal barrier and disrupt the mucosal protection mediated systemically by cyclooxygenase (COX) inhibition (Kuna et al., 2019).\u202f The patient\u2019s PUD may have been contributed by H.pylori infection, owing to the positive urease breath test, which reveals the presence of Helicobacter pylori bacteria.\u202f Besides, the PUD may have been caused by taking a high dose of ibuprofen 400-600 mg for pain relief.\u202f Ibuprofen causes reduced endogenous prostaglandins, resulting in local gastric mucosal injury.<\/span>\u00a0<\/span><\/p>\n In addition, lifestyle factors like tobacco smoking and excessive alcohol and caffeine consumption are associated with PUD.\u202f Caffeine stimulates the production of hydrochloric acid.\u202f Smoking accelerates gastric emptying and decreases pancreatic bicarbonate production (Kuna et al., 2019).\u202f Besides, ethanol irritates gastric mucosal and nonspecific gastritis.\u202f The patient\u2019s history of tobacco smoking, heavy caffeine intake, and daily alcohol intake may have led to the development of PUD.<\/span>\u00a0<\/span><\/p>\n \u202fKuna, L., Jakab, J., Smolic, R., Raguz-Lucic, N., Vcev, A., & Smolic, M. (2019). Peptic ulcer disease: a brief review of conventional therapy and herbal treatment options.\u202f<\/span>Journal of clinical medicine<\/span><\/i>,\u202f<\/span>8<\/span><\/i>(2), 179. doi: 10.3390\/jcm8020179<\/span>\u00a0<\/span><\/p>\n \u202f<\/span><\/p>\n A 65-year-old female comes to the clinic with a complaint of abdominal pain in the epigastric area. The pain has been persistent for two weeks. \u202fThe pain described as burning, non-radiating and worse after meals. Denies N&V, weight loss or obvious bleeding. She admits to frequent belching with bloating.\u202f\u202f<\/span>\u00a0<\/span><\/p>\n PMH:\u202f seasonal allergies with Chronic Sinusitis, positive for osteoarthritis,\u202f<\/span>\u00a0<\/span><\/p>\n Meds: Claritin 10 mg po daily, ibuprofen 400-600 mg po prn pain\u202f<\/span>\u00a0<\/span><\/p>\n Family Hx-non\u202fcontributary\u202f\u202f<\/span>\u00a0<\/span><\/p>\n Social history: Separated recently pending divorce; stressful situation with trying to manage two homes. Works as a Legal Assistant at a local law firm. She has 35 PPY of smoking, drinks 1-2 glasses of wine a day, and 6-7 cups of coffee per day. She\u202fdenies illicit drug use, vaping or unprotected sexual encounters.\u202f\u202f<\/span>\u00a0<\/span><\/p>\n Breath test in the office revealed + urease.\u202f<\/span>\u00a0<\/span><\/p>\n The healthcare provider suspects the client has peptic ulcer disease.<\/span>\u00a0<\/span><\/p>\n 1.<\/span><\/i>\u202f\u202f\u202f\u202f\u202f<\/span><\/i>What is the pathophysiology of PUD\/ formation of peptic ulcers?\u202f<\/span><\/i>\u00a0<\/span><\/p>\n \u00a0<\/span><\/p>\n Your Answer:<\/span>\u00a0<\/span><\/p>\n Peptic ulcers occur when there is a break in the mucous lining of the GI tract, and it comes into contact with hydrochloric acid and pepsin. The epithelial cells of the stomach and duodenum secrete mucus in response to irritation of the epithelial lining due to cholinergic stimulation. Ulcers or breaks in the mucosa of the GI tract occur with H. pylori infection, use of NSAIDs, trauma, infection, and physical or psychological stress (Alsinnari et al., 2022). H. pylori is spread by oral to oral, fecal-oral routes. It damages gastric epithelial cells reducing the effectiveness of gastric mucus. NSAIDs interrupt prostaglandin synthesis, which maintains the mucous barrier of the gastric mucosa. PUD can be chronic, with spontaneous remissions and exacerbations associated with trauma, infection, and physical or psychological stress.\u202f<\/span>\u00a0<\/span><\/p>\n Alsinnari, Y. M., Alqarni, M. S., Attar, M., Bukhari, Z. M., Almutairi, M., Baabbad, F. M., & Hasosah, M. (2022). Risk factors for recurrence of peptic ulcer disease: A retrospective study in tertiary care referral center.\u202f<\/span>Cureus<\/span><\/i>,\u202f<\/span>14<\/span><\/i>\n
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RESOURCES<\/span>\u00a0<\/span><\/h2>\n
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\nClick the weekly resources link to access the resources.\u202f<\/span>\u00a0<\/span><\/p>\nBY DAY 7 OF WEEK 5<\/span>\u00a0<\/span><\/h2>\n
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\n \u00a0<\/span><\/td>\n Attempt<\/span><\/b>\u00a0<\/span><\/td>\n Time<\/span><\/b>\u00a0<\/span><\/td>\n Score<\/span><\/b>\u00a0<\/span><\/td>\n<\/tr>\n \n LATEST<\/span><\/b>\u00a0<\/span><\/td>\n Attempt 1<\/span><\/a>\u00a0<\/span><\/td>\n 8,805 minutes<\/span>\u00a0<\/span><\/td>\n 18.7 out of 20<\/span>\u00a0<\/span><\/td>\n<\/tr>\n<\/tbody>\n<\/table>\n A Sample Answer For the Assignment: NURS 6501 GASTROINTESTINAL AND HEPATOBILIARY DISORDERS<\/strong><\/h2>\n
Title: NURS 6501 GASTROINTESTINAL AND HEPATOBILIARY DISORDERS<\/strong><\/h2>\n
Scenario 1: Peptic Ulcer<\/span><\/i><\/b>\u00a0<\/span><\/h2>\n
Questions:<\/span><\/b>\u00a0<\/span><\/h2>\n
References<\/span><\/b>\u00a0<\/span><\/h2>\n
A Sample Answer 2 For the Assignment: NURS 6501 GASTROINTESTINAL AND HEPATOBILIARY DISORDERS<\/strong><\/h2>\n
Title: NURS 6501 GASTROINTESTINAL AND HEPATOBILIARY DISORDERS<\/strong><\/h2>\n
Scenario 1: Peptic Ulcer<\/span><\/i><\/b>\u00a0<\/span><\/h2>\n
Question:<\/span><\/b>\u00a0<\/span><\/h2>\n
References<\/span>\u00a0<\/span><\/h2>\n