Sample Answer for NURS 6501 Discussion Endocrine Disorders<\/b>\u00a0Included After Question<\/strong><\/em><\/h2>\nIn this exercise, you will complete a 5-essay type question Knowledge Check to gauge your understanding of this module\u2019s content. \u202f<\/span>\u00a0<\/span><\/p>\nPossible topics covered in this Knowledge Check include:<\/span>\u00a0<\/span><\/p>\n\n- Diabetes<\/span>\u00a0<\/span><\/li>\n
- Hyper- and hypothyroidism<\/span>\u00a0<\/span><\/li>\n
- Adrenal disorders<\/span>\u00a0<\/span><\/li>\n
- Parathyroidism (hyper and hypo)<\/span>\u00a0<\/span><\/li>\n
- Checks & balances \/ negative feedback<\/span>\u00a0<\/span><\/li>\n
- Syndrome of Inappropriate Antidiuretic Hormone<\/span>\u00a0<\/span><\/li>\n
- Pheochromocytosis<\/span>\u00a0<\/span><\/li>\n
- Diabetes insipidus<\/span>\u00a0<\/span><\/li>\n
- Diabetic ketoacidosis<\/span>\u00a0<\/span><\/li>\n<\/ul>\n
Note<\/span><\/b>: It is strongly recommended that you take the Knowledge Check at least 48 hours before taking the Midterm Exam.<\/span>\u00a0<\/span><\/p>\n![\"Disorders\u00a0Included](\"http:\/\/nursingassignmentcrackers.com\/wp-content\/uploads\/2024\/01\/Capture-3-5-300x136.png\")
Disorders\u00a0Included After Question
NURS 6501 Discussion Endocrine Disorders<\/figcaption><\/figure>\nResources<\/span><\/b>\u00a0<\/span><\/h2>\nBe sure to review the Learning Resources before completing this activity. <\/span>
\nClick the weekly resources link to access the resources.\u202f<\/span>\u00a0<\/span><\/p>\nWEEKLY RESOURCES<\/span><\/b><\/a>\u00a0<\/span><\/p>\nBy Day 5 of Week 6<\/span><\/b>\u00a0<\/span><\/h2>\nComplete <\/span><\/b>the Knowledge Check by Day 5 of Week 6.<\/span>\u00a0<\/span><\/p>\nA Sample Answer For the Assignment:<\/strong>NURS 6501 Discussion Endocrine Disorders<\/span><\/b><\/strong><\/h2>\nTitle: <\/strong>NURS 6501 Discussion Endocrine Disorders<\/span><\/b><\/strong>\u00a0<\/span><\/h2>\nScenario 1: Syndrome of Antidiuretic Hormone (SIADH)<\/span><\/i><\/b>\u00a0<\/span><\/h3>\nA\u202f77-year-old\u202ffemale was brought to the clinic by her daughter who stated that her mother had become\u202fslightly\u202fconfused over the past several days. She had been stumbling at home and\u202fhad fallen twice but was able to walk with\u202fsome\u202fdifficulty. She had no other obvious problems and had been eating and drinking. The daughter became concerned when she forgot her daughter\u2019s name, so she thought she better bring her to the clinic.\u202f\u202f<\/span>\u00a0<\/span><\/p>\nHPI: Type II diabetes mellitus (DM) with peripheral neuropathy\u202fx 30 years.\u202fEmphysema. Situational depression after death of spouse 6-months\u202fago\u202f<\/span>\u00a0<\/span><\/p>\nSHFH:\u202f-\u202fnon contributary except for\u202f40 pack\/year history tobacco use.\u202f\u202f<\/span>\u00a0<\/span><\/p>\nMeds: Metformin 1000 mg po BID, ASA 81 mg po\u202fqam, escitalopram (Lexapro) 5 mg po q am\u202fstarted 2 months ago\u202f<\/span>\u00a0<\/span><\/p>\nLabs-CBC WNL;\u202fChem 7- Glucose-102 mg\/dl, BUN 16 mg\/dl, Creatinine 1.1 mg\/dl,\u202fNa<\/span>+<\/span>116 mmol\/L,\u202f<\/span>\u00a0<\/span><\/p>\nK<\/span>+<\/span>4.2 mmol\/L,\u202fCO<\/span>2<\/span>37 m\u202fmol\/L,\u202fCl<\/span>–<\/span>97 mmol\/L.\u202f\u202f<\/span>\u00a0<\/span><\/p>\nThe APRN refers the patient to the ED and called endocrinology for a consult for diagnosis and management of syndrome of inappropriate antidiuretic hormone\u202f(SIADH).\u202f<\/span>\u00a0<\/span><\/p>\nQuestion:<\/span><\/b>\u00a0<\/span><\/h2>\n1.<\/span><\/i>\u202f\u202f\u202f\u202f <\/span><\/i>Define SIADH and identify any patient characteristics that may have contributed to the development of SIADH\u202f<\/span><\/i>\u00a0<\/span><\/p>\n\u00a0<\/span><\/p>\nYour Answer:\u00a0<\/span>\u00a0<\/span><\/p>\nThis discussion question is about a 77-year-old female patient brought to the hospital by her daughter with complains that have led her being diagnosed with syndrome of inappropriate antidiuretic hormone. Syndrome of inappropriate antidiuretic hormone (SIADH) is a disorder characterized by the increased release of antidiuretic hormones. There is the inability to suppress the secretion of the ADH hormone leading to water toxicity. The kidneys cannot excrete excess water due to too much reabsorption, hence, hyponatremia. Patient factors contribute to the development of SIADH. One of them as seen in this case study is medications use. Some medications increase the patient\u2019s risk of developing SIADH when used for a long time. They include drugs such as antidepressants, seizure medications, diabetes, cancer, and hypertension treatment drugs(Kim, 2022). The patient in the case study is currently using drugs such as metformin and escitalopram, which have an elevated risk of SIADH.<\/span>\u00a0<\/span><\/p>\n\u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f Respiratory conditions such as acute respiratory failure, tuberculosis, and pneumonia also predispose patients to developing the disorder. The patient in the case study has a history of emphysema, which could precipitate other respiratory complications associated with SIADH. Disorders of the nervous system have also been linked with the development of SIADH in some patients. Accordingly, conditions such as head injury, tumors, stroke, and meningitis carry an increased risk of SIADH because of the involvement of the brain. The patient in the case study has neuropathy due to diabetes mellitus. In addition, she is at a risk of developing diabetes-related complication such as stroke. The fact that she experienced slight confusion and stumbling at home could indicate stroke (Mentrasti et al., 2020). Therefore, additional investigations should be conducted to rule out SIADH secondary to neurological conditions such as stroke.\u00a0<\/span>\u00a0<\/span><\/p>\nReferences<\/span><\/b>\u00a0<\/span><\/h2>\nKim, G.-H. (2022). Pathophysiology of Drug-Induced Hyponatremia. <\/span>Journal of Clinical Medicine<\/span><\/i>, <\/span>11<\/span><\/i>(19), Article 19. https:\/\/doi.org\/10.3390\/jcm11195810<\/a><\/span>\u00a0<\/span><\/p>\nMentrasti, G., Scortichini, L., Torniai, M., Giampieri, R., Morgese, F., Rinaldi, S., & Berardi, R. (2020). Syndrome of Inappropriate Antidiuretic Hormone Secretion (SIADH): Optimal Management. <\/span>Therapeutics and Clinical Risk Management<\/span><\/i>, <\/span>16<\/span><\/i>, 663\u2013672. https:\/\/doi.org\/10.2147\/TCRM.S206066<\/a><\/span>\u00a0<\/span><\/p>\nA Sample Answer 2 For the Assignment:<\/strong>NURS 6501 Discussion Endocrine Disorders<\/span><\/b><\/strong><\/h2>\nTitle: <\/strong>NURS 6501 Discussion Endocrine Disorders<\/span><\/b><\/strong><\/h2>\nScenario 2: Type 1 Diabetes<\/span><\/i><\/b>\u00a0<\/span><\/h3>\nA\u202f14-year-old\u202fgirl is brought to the pediatrician\u2019s office by his parents who are concerned about their daughter\u2019s weight loss\u202fdespite eating more,\u202ffrequent urination,\u202funquenchable thirst, and fatigue that is interfering with her school activities.\u202fShe had been seemingly\u202fhealthy until about 4 months ago when her parents started noticing these\u202fsymptoms. She admits to sleeping more\u202fand gets tired very easily.\u202f<\/span>\u00a0<\/span><\/p>\nPMH: noncontributory.\u00a0<\/span>\u00a0<\/span><\/p>\nAllergies-NKDA\u202f\u202f<\/span>\u00a0<\/span><\/p>\nFH:- maternal uncle with \u201csome kind of sugar diabetes problem\u201d but parents\u202funclear on the exact disease process\u202f<\/span>\u00a0<\/span><\/p>\nSH: denies alcohol, tobacco or illicit drug use. Not sexually active.\u202f<\/span>\u00a0<\/span><\/p>\nLabs:\u202frandom glucose 244 mg\/dl.\u202f\u202f<\/span>\u00a0<\/span><\/p>\nDIAGNOSIS: Diabetes Mellitus type 1 and refers to an endocrinologist for further work up and management plan.\u202f\u202f<\/span>\u00a0<\/span><\/p>\nQuestion<\/span><\/b>\u00a0<\/span><\/h2>\n1.<\/span><\/i>\u202f\u202f\u202f\u202f <\/span><\/i>Explain the pathophysiology of the three P\u2019s for (polyuria, polydipsia, polyphagia)\u201d with the given diagnosis of Type I DM.<\/span><\/i>\u00a0<\/span><\/p>\n\u00a0<\/span><\/p>\nYour Answer:\u00a0<\/span>\u00a0<\/span><\/p>\nThis discussion question is about a 14-year-old girl that has been brought to the pediatrician\u2019s office with complains of weight loss despite eating more, frequent urination, fatigue, and unquenchable thirst that has interfered with her school activities. The patient has been diagnosed with type 1 diabetes mellitus. Type 1 diabetes mellitus is a disorder characterized by the lack of insulin needed for the regulation of the normal blood glucose levels(Acharya et al., 2021). The patients present with symptoms that include polyuria, polydipsia, and polyphagia.<\/span>\u00a0<\/span><\/p>\n\u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f Polydipsia is an increase in thirst. Patients with type 1 diabetes mellitus often complain of an insatiable thirst. Patients with this disorder develop polydipsia because of the elevated blood glucose levels. The elevation results in the stimulation of the kidneys to excrete the excess glucose via the urine leading to glycosuria. On the other hand, the loss of fluids via the kidneys stimulates the brain to raise thirst level for the patient to take fluids to replace the loss (Del Chierico et al., 2022). Diabetes also causes osmotic diuresis, which is associated with dehydration and the increased need for body fluids.<\/span>\u00a0<\/span><\/p>\n\u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f Polyuria is the passage of urine more than normal. It is mostly more than 1-2 liters daily. As noted above an elevated blood glucose level results in the brain stimulating the kidneys to pass more urine, as a way of losing the excess glucose in it. The kidneys are stimulated to filter out more water, which increases the frequency, urgency, and volume of urine being passed. Polyphagia refers to excessive hunger. Patients with diabetes mellitus type I experience hunger because of glucose resistance. The glucose in the blood stream cannot enter the cells for metabolism due to the lack of insulin (Acharya et al., 2021). As a result, the cells continue being starved despite the high level of glucose, hence polyphagia.<\/span>\u00a0<\/span><\/p>\n\u202f<\/span><\/b>\u00a0<\/span>References<\/span><\/b>\u00a0<\/span><\/h2>\nAcharya, S., Shukla, S., & Vaswani, R. (2021). Pathophysiology of Complication in Diabetes Mellitus. <\/span>Journal of Pharmaceutical Research International<\/span><\/i>, 89\u201395. https:\/\/doi.org\/10.9734\/jpri\/2021\/v33i60A34459<\/a><\/span>\u00a0<\/span><\/p>\nDel Chierico, F., Rapini, N., Deodati, A., Matteoli, M. C., Cianfarani, S., &Putignani, L. (2022). Pathophysiology of Type 1 Diabetes and Gut Microbiota Role. <\/span>International Journal of Molecular Sciences<\/span><\/i>, <\/span>23<\/span><\/i>(23), Article 23. https:\/\/doi.org\/10.3390\/ijms232314650<\/a><\/span>\u00a0<\/span><\/p>\nA Sample Answer 3 For the Assignment:<\/strong>NURS 6501 Discussion Endocrine Disorders<\/span><\/b><\/strong><\/h2>\nTitle: <\/strong>NURS 6501 Discussion
Possible topics covered in this Knowledge Check include:<\/span>\u00a0<\/span><\/p>\n Note<\/span><\/b>: It is strongly recommended that you take the Knowledge Check at least 48 hours before taking the Midterm Exam.<\/span>\u00a0<\/span><\/p>\n Be sure to review the Learning Resources before completing this activity. <\/span> WEEKLY RESOURCES<\/span><\/b><\/a>\u00a0<\/span><\/p>\n Complete <\/span><\/b>the Knowledge Check by Day 5 of Week 6.<\/span>\u00a0<\/span><\/p>\n A\u202f77-year-old\u202ffemale was brought to the clinic by her daughter who stated that her mother had become\u202fslightly\u202fconfused over the past several days. She had been stumbling at home and\u202fhad fallen twice but was able to walk with\u202fsome\u202fdifficulty. She had no other obvious problems and had been eating and drinking. The daughter became concerned when she forgot her daughter\u2019s name, so she thought she better bring her to the clinic.\u202f\u202f<\/span>\u00a0<\/span><\/p>\n HPI: Type II diabetes mellitus (DM) with peripheral neuropathy\u202fx 30 years.\u202fEmphysema. Situational depression after death of spouse 6-months\u202fago\u202f<\/span>\u00a0<\/span><\/p>\n SHFH:\u202f-\u202fnon contributary except for\u202f40 pack\/year history tobacco use.\u202f\u202f<\/span>\u00a0<\/span><\/p>\n Meds: Metformin 1000 mg po BID, ASA 81 mg po\u202fqam, escitalopram (Lexapro) 5 mg po q am\u202fstarted 2 months ago\u202f<\/span>\u00a0<\/span><\/p>\n Labs-CBC WNL;\u202fChem 7- Glucose-102 mg\/dl, BUN 16 mg\/dl, Creatinine 1.1 mg\/dl,\u202fNa<\/span>+<\/span>116 mmol\/L,\u202f<\/span>\u00a0<\/span><\/p>\n K<\/span>+<\/span>4.2 mmol\/L,\u202fCO<\/span>2<\/span>37 m\u202fmol\/L,\u202fCl<\/span>–<\/span>97 mmol\/L.\u202f\u202f<\/span>\u00a0<\/span><\/p>\n The APRN refers the patient to the ED and called endocrinology for a consult for diagnosis and management of syndrome of inappropriate antidiuretic hormone\u202f(SIADH).\u202f<\/span>\u00a0<\/span><\/p>\n 1.<\/span><\/i>\u202f\u202f\u202f\u202f <\/span><\/i>Define SIADH and identify any patient characteristics that may have contributed to the development of SIADH\u202f<\/span><\/i>\u00a0<\/span><\/p>\n \u00a0<\/span><\/p>\n Your Answer:\u00a0<\/span>\u00a0<\/span><\/p>\n This discussion question is about a 77-year-old female patient brought to the hospital by her daughter with complains that have led her being diagnosed with syndrome of inappropriate antidiuretic hormone. Syndrome of inappropriate antidiuretic hormone (SIADH) is a disorder characterized by the increased release of antidiuretic hormones. There is the inability to suppress the secretion of the ADH hormone leading to water toxicity. The kidneys cannot excrete excess water due to too much reabsorption, hence, hyponatremia. Patient factors contribute to the development of SIADH. One of them as seen in this case study is medications use. Some medications increase the patient\u2019s risk of developing SIADH when used for a long time. They include drugs such as antidepressants, seizure medications, diabetes, cancer, and hypertension treatment drugs(Kim, 2022). The patient in the case study is currently using drugs such as metformin and escitalopram, which have an elevated risk of SIADH.<\/span>\u00a0<\/span><\/p>\n \u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f Respiratory conditions such as acute respiratory failure, tuberculosis, and pneumonia also predispose patients to developing the disorder. The patient in the case study has a history of emphysema, which could precipitate other respiratory complications associated with SIADH. Disorders of the nervous system have also been linked with the development of SIADH in some patients. Accordingly, conditions such as head injury, tumors, stroke, and meningitis carry an increased risk of SIADH because of the involvement of the brain. The patient in the case study has neuropathy due to diabetes mellitus. In addition, she is at a risk of developing diabetes-related complication such as stroke. The fact that she experienced slight confusion and stumbling at home could indicate stroke (Mentrasti et al., 2020). Therefore, additional investigations should be conducted to rule out SIADH secondary to neurological conditions such as stroke.\u00a0<\/span>\u00a0<\/span><\/p>\n Kim, G.-H. (2022). Pathophysiology of Drug-Induced Hyponatremia. <\/span>Journal of Clinical Medicine<\/span><\/i>, <\/span>11<\/span><\/i>(19), Article 19. https:\/\/doi.org\/10.3390\/jcm11195810<\/a><\/span>\u00a0<\/span><\/p>\n Mentrasti, G., Scortichini, L., Torniai, M., Giampieri, R., Morgese, F., Rinaldi, S., & Berardi, R. (2020). Syndrome of Inappropriate Antidiuretic Hormone Secretion (SIADH): Optimal Management. <\/span>Therapeutics and Clinical Risk Management<\/span><\/i>, <\/span>16<\/span><\/i>, 663\u2013672. https:\/\/doi.org\/10.2147\/TCRM.S206066<\/a><\/span>\u00a0<\/span><\/p>\n A\u202f14-year-old\u202fgirl is brought to the pediatrician\u2019s office by his parents who are concerned about their daughter\u2019s weight loss\u202fdespite eating more,\u202ffrequent urination,\u202funquenchable thirst, and fatigue that is interfering with her school activities.\u202fShe had been seemingly\u202fhealthy until about 4 months ago when her parents started noticing these\u202fsymptoms. She admits to sleeping more\u202fand gets tired very easily.\u202f<\/span>\u00a0<\/span><\/p>\n PMH: noncontributory.\u00a0<\/span>\u00a0<\/span><\/p>\n Allergies-NKDA\u202f\u202f<\/span>\u00a0<\/span><\/p>\n FH:- maternal uncle with \u201csome kind of sugar diabetes problem\u201d but parents\u202funclear on the exact disease process\u202f<\/span>\u00a0<\/span><\/p>\n SH: denies alcohol, tobacco or illicit drug use. Not sexually active.\u202f<\/span>\u00a0<\/span><\/p>\n Labs:\u202frandom glucose 244 mg\/dl.\u202f\u202f<\/span>\u00a0<\/span><\/p>\n DIAGNOSIS: Diabetes Mellitus type 1 and refers to an endocrinologist for further work up and management plan.\u202f\u202f<\/span>\u00a0<\/span><\/p>\n 1.<\/span><\/i>\u202f\u202f\u202f\u202f <\/span><\/i>Explain the pathophysiology of the three P\u2019s for (polyuria, polydipsia, polyphagia)\u201d with the given diagnosis of Type I DM.<\/span><\/i>\u00a0<\/span><\/p>\n \u00a0<\/span><\/p>\n Your Answer:\u00a0<\/span>\u00a0<\/span><\/p>\n This discussion question is about a 14-year-old girl that has been brought to the pediatrician\u2019s office with complains of weight loss despite eating more, frequent urination, fatigue, and unquenchable thirst that has interfered with her school activities. The patient has been diagnosed with type 1 diabetes mellitus. Type 1 diabetes mellitus is a disorder characterized by the lack of insulin needed for the regulation of the normal blood glucose levels(Acharya et al., 2021). The patients present with symptoms that include polyuria, polydipsia, and polyphagia.<\/span>\u00a0<\/span><\/p>\n \u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f Polydipsia is an increase in thirst. Patients with type 1 diabetes mellitus often complain of an insatiable thirst. Patients with this disorder develop polydipsia because of the elevated blood glucose levels. The elevation results in the stimulation of the kidneys to excrete the excess glucose via the urine leading to glycosuria. On the other hand, the loss of fluids via the kidneys stimulates the brain to raise thirst level for the patient to take fluids to replace the loss (Del Chierico et al., 2022). Diabetes also causes osmotic diuresis, which is associated with dehydration and the increased need for body fluids.<\/span>\u00a0<\/span><\/p>\n \u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f\u202f Polyuria is the passage of urine more than normal. It is mostly more than 1-2 liters daily. As noted above an elevated blood glucose level results in the brain stimulating the kidneys to pass more urine, as a way of losing the excess glucose in it. The kidneys are stimulated to filter out more water, which increases the frequency, urgency, and volume of urine being passed. Polyphagia refers to excessive hunger. Patients with diabetes mellitus type I experience hunger because of glucose resistance. The glucose in the blood stream cannot enter the cells for metabolism due to the lack of insulin (Acharya et al., 2021). As a result, the cells continue being starved despite the high level of glucose, hence polyphagia.<\/span>\u00a0<\/span><\/p>\n Acharya, S., Shukla, S., & Vaswani, R. (2021). Pathophysiology of Complication in Diabetes Mellitus. <\/span>Journal of Pharmaceutical Research International<\/span><\/i>, 89\u201395. https:\/\/doi.org\/10.9734\/jpri\/2021\/v33i60A34459<\/a><\/span>\u00a0<\/span><\/p>\n Del Chierico, F., Rapini, N., Deodati, A., Matteoli, M. C., Cianfarani, S., &Putignani, L. (2022). Pathophysiology of Type 1 Diabetes and Gut Microbiota Role. <\/span>International Journal of Molecular Sciences<\/span><\/i>, <\/span>23<\/span><\/i>(23), Article 23. https:\/\/doi.org\/10.3390\/ijms232314650<\/a><\/span>\u00a0<\/span><\/p>\n\n
NURS 6501 Discussion Endocrine Disorders<\/figcaption><\/figure>\nResources<\/span><\/b>\u00a0<\/span><\/h2>\n
\nClick the weekly resources link to access the resources.\u202f<\/span>\u00a0<\/span><\/p>\nBy Day 5 of Week 6<\/span><\/b>\u00a0<\/span><\/h2>\n
A Sample Answer For the Assignment:<\/strong>NURS 6501 Discussion Endocrine Disorders<\/span><\/b><\/strong><\/h2>\n
Title: <\/strong>NURS 6501 Discussion Endocrine Disorders<\/span><\/b><\/strong>\u00a0<\/span><\/h2>\n
Scenario 1: Syndrome of Antidiuretic Hormone (SIADH)<\/span><\/i><\/b>\u00a0<\/span><\/h3>\n
Question:<\/span><\/b>\u00a0<\/span><\/h2>\n
References<\/span><\/b>\u00a0<\/span><\/h2>\n
A Sample Answer 2 For the Assignment:<\/strong>NURS 6501 Discussion Endocrine Disorders<\/span><\/b><\/strong><\/h2>\n
Title: <\/strong>NURS 6501 Discussion Endocrine Disorders<\/span><\/b><\/strong><\/h2>\n
Scenario 2: Type 1 Diabetes<\/span><\/i><\/b>\u00a0<\/span><\/h3>\n
Question<\/span><\/b>\u00a0<\/span><\/h2>\n
\u202f<\/span><\/b>\u00a0<\/span>References<\/span><\/b>\u00a0<\/span><\/h2>\n
A Sample Answer 3 For the Assignment:<\/strong>NURS 6501 Discussion Endocrine Disorders<\/span><\/b><\/strong><\/h2>\n
Title: <\/strong>NURS 6501 Discussion